Ignite Creation Date:
2024-05-06 @ 3:49 PM
Last Modification Date:
2024-10-26 @ 1:57 PM
Study NCT ID:
NCT04760600
Status:
UNKNOWN
Last Update Posted:
2021-02-18
First Post:
2021-02-05
Brief Title:
Role of Parasitic Infection in Inflammatory Bowel Disease and Irritable Bowel Syndrome
Sponsor:
Assiut University
Organization:
Assiut University
Study Overview
Official Title:
Role of Parasitic Infection in Inflammatory Bowel Disease and Irritable Bowel Syndrome
Status:
UNKNOWN
Status Verified Date:
2021-01
Last Known Status:
RECRUITING
Delayed Posting:
No
If Stopped, Why?:
Not Stopped
Has Expanded Access:
False
If Expanded Access, NCT#:
N/A
Has Expanded Access, NCT# Status:
N/A
Acronym:
None
Brief Summary:
Role of parasitic infection in Inflammatory bowel disease and Irritable bowel The burden of the parasitic disease is in-direct relation with the health and nutritional status of the host inducing not only parasite-specific immunity but also can modify the hosts immune responses Many parasites can imitate inflammatory bowel disorders and some studies showed that infection with parasites can improve disorders like IBD or moderate the symptoms of inflammatory bowel disorders Even though many studies have shown that gastrointestinal infection is an important risk factor for the development of IBS
Detailed Description:
1 Detect the prevalence of parasites associated with Inflammatory bowel diseases IBD and Irritable bowel syndrome IBS in comparison with control subjects
2 Evaluating the effect of the parasite on the course and prognosis of the diseases
3 Evaluating the effect of the antiparasitic drugs on the course and prognosis of the diseases
2 Evaluating the effect of the parasite on the course and prognosis of the diseases
3 Evaluating the effect of the antiparasitic drugs on the course and prognosis of the diseases
Study Oversight
Has Oversight DMC:
None
Is a FDA Regulated Drug?:
False
Is a FDA Regulated Device?:
False
Is an Unapproved Device?:
None
Is a PPSD?:
None
Is a US Export?:
None
Is an FDA AA801 Violation?:
None